Fructose and Sugar: A Major Mediator of Nonalcoholic Fatty Liver Disease - PMC
Thomas Jensenncbi.nlm.nih.govSaved by Seth Kramer
Fructose and Sugar: A Major Mediator of Nonalcoholic Fatty Liver Disease - PMC
Saved by Seth Kramer
there is one single cause of accelerated aging and diseases of aging, it is this: sugar and starch (especially flour) and the resulting metabolic chaos and insulin resistance.
“If only a small fraction of what is already known about the effects of sugar were revealed in relation to any other material used as a food additive,” Yudkin wrote, “that material would promptly be banned.”34
It’s almost as if you have a bathtub, and you’re filling it up from the faucet. If you keep the faucet running even after the tub is full and the drain is closed (i.e., you’re sedentary), water begins spilling over the rim of the tub, flowing into places where it’s not wanted or needed, like onto the bathroom floor, into the heating vents or down t
... See moreHumans store extra glucose as glycogen and fat. Extra fructose just turns into fat.
It also states that greater consumption of SFA was linked to slower generation of atherosclerosis, while PUFA and Carb intake (most likely carb-heavy foods combined with high PUFA concentration) were linked to an increased progression.
first, it accumulates in the liver and drives the development of nonalcoholic fatty liver disease.