How Our Hormones Control Our Hunger, Eating & Satiety

There are endokrine signal involved to signal hunger and satiety. The archuit nucleus release chemicals that act as brakes and accelerators of hunger. - The PMOC neurons (system) produce a-MSH, which reduces appetite and get active while eating. They are activated by ultra-violate light to the eyes, which explains the lower hunger and appetite in t... See more

Ventral medial hypothalamus: control station for hunger, eating and satiety through multiple populations of neurons, which respectively control hunger and satiety.

Ghrelin is released from the GI tract and stimulates your desire to eat. It is triggered by low glucose levels in the blood and triggers anticipatory food signals making you think about food you would like to eat. Your ghrelin secretion will start to match your eating schedule. It can be shifted by about 45 minutes a day.

When you are hungry you secrete glucagon, which extracts stores of energy out of the liver and the muscles. When these are depleted you use body fat. Consuming carbohydrates or fats first causes a spike in glucose often triggering you eat more. If you eat the fibrous things first, it blunts the increase of glucose and the increase of glucose of sub... See more

HIIT and resistance training promotes cycling glucose into glucogen, while steady state cardio promotes stable glucose levels. Urin is filtered blood.

You should avoid sharp spikes in glucose. Movement before a meal can dumpen the glucose response.

Glutamine also reduces sugar craving by increasing blood sugar levels. Emulsifiers strip away the mucosal lining of the blood and cause the neurons to retract deeper into the tract. This stops the triggering of CCK signals. They also increase the neurons measuring sugar and trigger dopamine in the brain. This can lead to an overcompensation effect ... See more

CCK is released from the GI tract when we digest fatty acids (Omega 3, 6), amino acids (especially glutamine) and sugar. We mostly eat until we consumed enough fatty acids and amino acids to trigger CCK. CCK then puts the break on a-GRP neurons.

Insulin’s shuttles glucose to the different parts of the body to be used as fuel. Your blood sugar needs to be kept in a certain range. If glucose levels get to high, the glucose can damage and destroy neurons.